Can hookworm hook you?
“Worms” are everywhere. Probably the
commonest infestation of mankind. All children go through harboring worms at
some stage, but usually the tiny thread worms.
Of course, when you talk about ‘worms’,
most people only think of threadworms that all children seem to get. The
answer is always “a good worming”. Yes, we’ve all been down to the chemist
shop and bought a packet of ‘worming’ tablets.
However, you should not imagine that
‘worms’ stops there. I can assure you that there are far more dangerous
wrigglers in the community. And the sandy beaches are the habitat for some
My friends laugh at me when they see me
walking along the beach. Instead of letting the cool sand squish between my
toes, I wear closed shoes. Does this mean I am a pedantic pedestrian? Or a
member of a weird anti-sandal sect? Fortunately it is neither. I am just a
trifle afraid of Ancylostoma duodenale. And so should you!
Ancylostoma duodenale is one of the two
hookworms that can get their hooks into you (and me if I let them). The
other is called Necator americanus. These little chaps are roundworms
between 7 to 13 mm long and are far from rare. Approximately one-quarter of
the world's population is infected with Hookworm.
So how do you get infected? Easy, the
hookworm eggs are passed in feces (or poop if you prefer) and infection
results when you come in contact with the eggs from the contaminated soil.
The larvae enter through the skin and travel to the lungs through the blood.
They ascend the lungs through the bronchi and trachea and are then
swallowed. As the larvae pass into the digestive tract, they attach
themselves to the wall of the small intestine. Here they mature into adult
worms, mate and feed on the blood of the host. And adult hookworms may live
up to ten years. A not so Happy Birthday!
Unfortunately many hookworm
infestations do not produce symptoms; however, there may be local irritation
of the skin where the worm penetrated or even an itchy rash. While going
through the lungs, there may be asthma-like symptoms or even pneumonia. The
most common symptoms of hookworm infection, however, are from their taking
up residence in the intestine. Hookworm here can lead to abdominal pain,
diarrhea, weight loss, loss of appetite and excessive gas.
With long-standing infections, the
intestine’s owner may become anemic as the worms feed on the individual's
blood. This in turn leads to the usual anemic symptoms including pale
complexion, tiredness and weakness.
Diagnosis is done by looking for
hookworm eggs in the stool (by using a microscope). Blood tests will show
the amount of blood loss and can be used as a pointer towards the
seriousness of the infestation.
Fortunately hookworm is treatable,
generally with the drug mebendazole. This drug cures more than 99 percent of
all cases of hookworm if given twice per day for three days. It kills both
the worms and the eggs, but is contraindicated during pregnancy (your
pregnancy, not its pregnancy!). If anemia has become a problem, then iron
supplements can be given as well. Once treated, the symptoms settle quickly
in a few weeks at most.
So what can you do to avoid hookworms?
Well since hookworm infection comes from non-hygienic practices and fecal
contact in the soil, my shoes sound like a great idea, don’t you think?
Never mind the problems with hypodermic needles found in the sand in many
countries these days!
Hookworm infections should be dealt
quickly and stringently. Known symptomatic infections should be treated
rapidly and treatment given to asymptomatic family members or neighbors.
Strict attention to cleanliness and sanitary practices is needed when a
hookworm infection is detected to prevent its spread. This means hygienic
disposal of human waste, limiting skin contact with soil and even water,
where there is untreated sewage.
Hookworm can be a very serious illness
so it is essential to be aware of any change in one's health status. Any
difficulty breathing, rapid heartbeat, chest or abdominal pain, bloody
diarrhea, blood with coughing, asthma-like symptoms, skin rashes, abdominal
swelling or bloating, lightheadedness or weight loss should be brought to
your doctor's attention.
Me? I’ll just keep wearing shoes!
Why you should stay in touch with the family
We all have much to thank our family
for. Just letting us grow up for starters. As an aside, if your young
children are demanding attention with terrible two year old tantrums, they
do grow out of them. My young son was lucky to reach his third birthday, but
his mother shielded him from paternal wrath and he has grown up to be a nice
young 11 year old.
However, the family heredity is one of
the ‘clues’ to your health in the future, and what you can do to enjoy a
long, lively and healthy one. This is where ‘thanks Mum and Dad’ comes in.
One problem of being an orphan is that
it leaves the person with no idea as to what hereditary ailments are going
to befall them. Dad might have legged it (an Olympic sport in Thailand) or
‘fled the scene’, but did he live to tell the tale when he was 60 when he
suffered from heart disease?
With the increasing research into
genetics, we are able to map out our likely futures and can predict such
ailments as diabetes, epilepsy and other neurological problems like
Huntington’s Chorea and Alzheimer’s Disease, some cancers such as breast,
ovarian, lower bowel, prostate, skin and testicular, heart attacks, blood
pressure problems, certain blood diseases like Sickle Cell anemia and so the
list goes on.
However, you do not need to have
multi-million baht examinations done on your DNA to see where you are
headed, all you need to do is to start asking the older family members about
your inheritance. Not the money - your genetic inheritance in the health
Have you ever wondered why the
questionnaire for life insurance asks whether any close member of your
family has ever suffered from diabetes, epilepsy and other ailments and then
also asks you to write down how old your parents or brothers and sisters
were when they died, and what they died from? All that they, the insurance
companies, are doing is finding out the relative likelihood (or ‘risk’) of
your succumbing early to an easily identifiable hereditary condition. This
does not need a postgraduate Masters degree in rocket science. It only needs
a cursory application of family history.
If either of your parents had diabetes,
your elder brother has diabetes, your younger brother has diabetes and your
cousin has diabetes, what are the odds on your getting (or already having)
diabetes? Again this is not rocket science. The answer is pretty damn high!
And yet, I see families like this, where the individual members are totally
surprised and amazed when they fall ill, go to hospital, and diabetes is
It does not really take very much time
over a family lunch to begin to enquire about one’s forebears. After five
minutes it will be obvious if there is some kind of common medical thread
running through your family. That thread may not necessarily be life
threatening, but could be something like arthritis for example.
Look at it this way - your future is
being displayed by your family’s past. This could be considered frightening,
when your father, his brother and your grandfather all died very early from
heart attacks. Or, this could be considered as life saving, if it pushes you
towards looking at you own cardiac health and overcoming an apparently
disastrous medical history.
This is the advantage that you get
provided you are not an orphan. You know what to look for before it becomes
a problem. Going back to the family with diabetes, what should the younger
members do? Well, if it were me, I would be having my blood sugar checked at
least once a year from the age of 20. Any time I had reason to visit the
doctor in between, I would also ask to have the level checked. We are
talking about a very inexpensive test that could literally save you millions
of baht in the future, as well as giving you a better quality of life, and a
Ask around the dinner table today and
plan to check your medical future tomorrow. It’s called a ‘Check-up’!
Genghis Khan and your children
Some medical facts: Bruises on a
child’s body are often considered proof that a baby has been battered. A
visible bruise on the buttocks, the shape of a hand and five fingers is
almost ‘undeniable’ proof. In fact, there was a very celebrated instance of
a GP in the UK having discovered that so many of the Asian babies in the
practice were showing signs of being ‘battered’ that the children’s welfare
people were called in and an enormous number of children taken away.
However, the highly observant GP was wrong!
In Thailand, and the rest of Asia, a
new-born baby with the ‘handprint’ bruise is very common, while child abuse
is not common at all. The problem, or rather the condition, relates back to
Genghis Khan and the Mongol hordes. It is a wonderful piece of folklore and
also a fine example of applied genetics.
Let’s look at the folklore first, and
you are going to have to dig very deep to get this tale anywhere else! A
Mongolian baby, called Tanujin, was born just over 1,000 years ago, but did
not breathe. His father, in desperation, held his new-born son upside down
and smacked him severely over the bottom, so much so that the baby drew
breath and lived, but carried the life giving bruise for the rest of his
days. That baby later became Genghis Khan, (which means King of the Earth),
and by the time he died in 1227 he was the ruler of a large chunk of it,
including the area which later became known as Thailand.
History has chronicled that the Mongol
hordes raped, pillaged and annexed countries from China to Persia. His
highly mobile troops traveled the difficult terrain of Siberia. Famous
cities were captured and looted such as Tashkent, Baghdad (still a good
place to stay away from, thanks George) and Bokhara. Cities that surrendered
were spared but those that resisted were razed and the people slaughtered.
The Mongols conquered northern India and Afghanistan. In 1222, they defeated
the Russian and Bulgarian armies. At the time of Genghis Khan’s death, his
empire stretched from China’s Yellow River to the Dnieper, in Russia.
And now back to some interesting
folklore. The descendants of Genghis Khan also showed the hand-shaped bruise
on the buttocks, beginning with his four sons Ogdai, Jagatai, Juji and Tule,
who were given one quarter of the empire each after their father died. They
in turn passed on this ‘trademark’ and so this continues till today. If your
“Luk Krung” children have the sign of Genghis Khan, called Mongolian Blue
Spot, you can claim descent from the warrior king. However, there is quite a
number of you, so I think there won’t be much left in Genghis’ estate by
Now Mongolian Blue Spot, as a clinical
condition, is well documented, and I came across figures suggesting that at
least one Mongolian spot is present on over 90 percent of Native Americans
and people of African descent, over 80 percent of Asians, over 70 percent of
Hispanics, and just under 10 percent of fair-skinned infants (Clinical
Pediatric Dermatology, 1993).
Medically we describe Mongolian Blue
Spot as flat bluish to bluish gray skin markings that commonly appear at
birth (or shortly thereafter) and scientifically they are called congenital
dermal melanocytosis. They are flat, pigmented lesions with nebulous borders
and irregular shape. They appear commonly at the base of the spine, on the
buttocks and back, but also can appear as high as the shoulders and
elsewhere. The medical text books also warn that occasionally Mongolian Blue
Spots are mistaken for bruises and questions about child abuse arise.
Obviously a text book that the UK GP did not read! Mongolian Blue Spots are
birthmarks, not bruises.
So, for all of you with children with a
peculiar blue birthmark on their bottoms, or for those interested in
checking friends and neighbors (or the young ladies dancing in the chrome
pole palaces), it seems fairly positive that the lineage is verified. You
really have found descendants of the man who conquered more of the world
than Alexander the Great. And guess what – my children have it too!
Biting on a bullet!
Surgeons can be the ‘prima donnas’ of
medicine, if you like. It is the surgeons who get the headlines in the
newspapers. It is the surgeons who are the stars in movies and TV. Who can
remember the irascible surgeon Sir Lancelot Spratt (Dr. In The House, 1954)
or the young surgeon Dr. Kildare (1961)? Slightly more recent, the American
surgeons in M*A*S*H?
However, surgeons have been around for
many centuries and have their own Royal College. The origins of the first
Royal College of Surgeons go back to the fourteenth century with the
foundation of the 'Guild of Surgeons Within the City of London'. There was
dispute between the surgeons and barber surgeons until an agreement was
signed between them in 1493, giving the fellowship of surgeons the power of
incorporation. In 1745 the surgeons broke away from the barbers to form the
Company of Surgeons. In 1800 the Company was granted a Royal Charter to
become the Royal College of Surgeons in London. A further charter in 1843
granted it the present title of the Royal College of Surgeons of England (of
which I proudly say I am a member).
We marvel at the surgical advances in
the past century, but while I take my hat off to the surgeons, the real
praise goes to the anesthetists. Without the advances in anesthetics, brawny
assistants would still be holding patients down while surgeons attacked with
scalpels and saws and the patient lay there biting on a bullet.
The first anesthetic agent was ether,
dribbled on to a mask to knock the patient out and allow the surgeon to take
his time and become meticulous in his approach. The first public
demonstration of ether anesthesia took place on 16 October 1846, at
Massachusetts General Hospital in Boston. The anesthetist was William Morton
and the surgeon was John Warren; and the operation was the removal of a lump
under the jaw of a Gilbert Abbott.
While there have been enormous advances
since then, I can remember being a medical student and assisting at an
operation in outback Australia in 1964. The anesthetic was ether, dribbled
on to the patient’s gauze mask by the matron of the public hospital, and it
was a Caesarian section for twins. There was no air-conditioning and it was
43 degrees in the theatre, where the fumes were making us all woozy.
Amazingly everyone survived the ordeal, mother, twin sons, the local doctor,
the matron and me.
Despite outback Australia, anesthesia
progressed in the rest of the world. Chloroform was introduced by James
Simpson, the Professor of Obstetrics in Edinburgh, in November 1847. This
was a more potent agent but it had more severe side effects, including
sudden death. However, it worked well and was easier to use than ether and
so, despite its drawbacks, became very popular.
The next major advance was the
introduction of local anesthesia – cocaine – in 1877. Things definitely did
go better with ‘coke’! Then came local infiltration, nerve blocks and then
spinal and epidural anesthesia, which in the 1900s allowed surgery in a
relaxed abdomen, and is still used today, especially in obstetric
anesthesia, where the mother can be anaesthetized without the baby being
affected as well.
The next important innovation was the
control of the airways with the use of tubes placed into the trachea. This
permitted control of breathing and techniques introduced in the 1910s were
perfected in the late 1920s and early 1930s. Then came the introduction of
intravenous induction agents. These were barbiturates which enabled the
patient to go off to sleep quickly, smoothly and pleasantly and therefore
avoided any unpleasant inhalational agents. Then in the 1940s and early
1950s, there came the introduction of muscle relaxants, firstly with curare
(the South American Indian poison, but not administered by native blowpipe)
and then agents less dangerous.
Anesthesia is now very safe, with
mortality of less than 1 in 250,000 directly related to anesthesia.
Nevertheless, with today’s sophisticated monitoring systems and a greater
understanding of bodily functions, the anesthetic profession will continue
to strive for improvement over the next 150 years.
On behalf of all patients requiring
surgery in the future I thank the anesthetists. No longer do they have to
bite on this bullet!
The diced carrot syndrome
For many of my childhood years I
wondered why every time I vomited, it looked as if the vomitus contained
diced carrot. True? You have found the same. Even when you haven’t eaten
diced carrot for several months.
I also thought that my stomach was like
a tray with compartments, with one for potatoes, another for cabbage, etc.
How did the food know which compartment to dive into? My young mind worked
that one out as well – it was the uvula (the clapper thingy at the back of
the throat) that kicked the potatoes in the correct direction. No wonder I
ended up studying medicine at university.
However, minds greater than mine (who
actually know about the uvula) have announced to the world that a
dinosaur-like animal that looked like a dolphin and swam like a fish can add
another string to its bow – it was frequently as sick as a parrot.
Scientists have discovered the oldest
fossilized vomit of ichthyosaurs, (an ancient marine reptile that lived 160
million years ago) which delighted in feeding off squid-like prey which had
indigestible shells. For those who are worried, you are not likely to
accidentally step on fresh ichthyosaur vomit as it is fossilized vomit that
has the researchers all excited.
The scientists unearthed the
regurgitated stomach contents of ichthyosaurs in a clay quarry near
Peterborough, north of London, which has provided the researchers with a
fascinating insight into the feeding habits of these long-extinct creatures.
“We believe that this is the first time
the existence of fossil vomit on a grand scale has been proven beyond
reasonable doubt,” said Peter Doyle, professor of geoscience from the
University of Greenwich.
“It seems that ichthyosaurs regularly
regurgitated the harder elements of its meal, rather like an owl coughs up a
pellet of indigestible bones and fur after digesting its prey. (I knew
there was a reason I have never trusted owls.)
“The vomitus, known as “splat” contains
the distinctive shells of belemnites, the nutritious shellfish on which the
ichthyosaurs fed, which 160 million years later have been partly only
digested by the reptile's gastric juices.
“The Peterborough belemnite shells,
viewed under a powerful electron microscope, have revealed acid-etching
marks caused by the digestive fluids from the gut of the marine reptile,”
Professor Doyle said.
“This proves that the belemnites had
been eaten by a predator. The fact that most of these belemnites were
juveniles, reinforces our view that the belemnites did not die of old age,”
he said. The plot thickens. (Or is that the splat thickens?)
It seems that Ichthyosaurs were to
ancient reptiles what dolphins and whales are to mammals today, an animal
perfectly adapted to a fully marine life which evolved from a terrestrial
ancestor, and not the other way around as popular comic book ‘science’ would
have you believe.
Professor Doyle, who made the discovery
with Jason Wood of the Open University, said the fossil vomit clears up a
long-standing mystery of what happened to the shells of its belemnite diet.
“It is highly unlikely that these
shells passed through the ichthyosaur's intestines and were excreted as
droppings, as they would have damaged the soft tissue of the reptile's
internal organs,” Professor Doyle said.
“The only scientific alternative is
that the shells were vomited out, in much the same way that modern-day sperm
whales regurgitate the indigestible beaks of squid they have eaten,” he
As an adjunct to the ichthyosaur story,
it has been postulated that the ichthyosaurs and plesiosaurs co-existed, and
the ichthyosaurs were preyed upon by the much larger plesiosaurs, thus
causing the smaller animal to ingest prehistoric squid.
And to bring the item right up to date,
Angela Milner, associate keeper of paleontology at the Natural History
Museum in London, says this idea (of having stones in the plesiosaur’s
stomach) makes sense. “I don't think it has been suggested before that
(gastroliths) might have acted as a gastric mill, but there is no real
reason why not,” she said. All that just to eat the belemnites.
And to throw the Xenosmilus
(prehistoric cats) amongst the pterodactyls, the Loch Ness Monster was a
Fractured collar bones
Fracture is the medical term for a
crack or a break in the structure of a bone. There are many different types
of fractures including:
Open or compound fracture, where the
fracture site is open to air because one end of the bone has broken through
Closed fracture: neither end of the
bone has broken through the skin.
Complete fracture: the broken bone is
completely separated at the break.
Incomplete fracture: the broken bone is
not completely separated at the break.
Transverse fracture: a straight break
across the bone.
Spiral fracture or oblique fracture:
usually caused by sudden, violent, rotating movements, such as twisting the
leg during a fall.
Comminuted fracture: there are more
than two fragments of bone at the fracture site.
Compression fracture: the break occurs
because of extreme pressure on the bone.
Impacted fracture: the broken ends are
driven into each other.
Avulsion fracture: the breaking force
has been applied in such a way that the muscle pulls a portion of the bone
away from the site where it is normally attached.
Pathological fracture: the fracture
occurs in a bone that is weakened or damaged by disease.
Torus fracture or a greenstick or
ripple fracture: on one side of the bone. Always a children’s fracture and
Stress fracture: microscopic fractures
caused by repeated jarring and overuse of a bone. This is typically seen in
Fractures can be displaced or not
displaced. A displaced fracture means the bone has shifted its position
relative to the bone on the other side of the fracture.
What is a Fracture of the Clavicle
(“Collar Bone”)? Your clavicle bone or “collar bone” connects the scapula
bone in your shoulder to your sternum in your chest. Its function is to hold
the shoulder upward and backward.
Clavicle fractures are among the most
common bone injuries. A break in the clavicle bone is usually always a
closed fracture that normally takes about 6 weeks to heal in an adult, 4
weeks in a child.
Surgery is rarely needed.
Causes of a Fracture of the Clavicle?
At the time of birth, the clavicle may
fracture during passage through the birth canal. The fracture is frequently
not diagnosed until the healing bone callus is noticed as a hard lump. At
this time it needs no treatment and the lump will disappear as the baby
Accidents such as falls against the
shoulder or on an outstretched hand are the most common cause of fractures
of the clavicle.
Sometimes, a blow from a blunt object
or a collision of some sort can cause the clavicle to break.
Symptoms of a Fracture of the Clavicle
are the same for almost all fractures.
Deformity or “bump” at the site of the
If asked to lift their arm, patients
with a broken clavicle cannot do so without extreme pain.
Treatment of a Fracture of the
The goal of treating broken bones is to
set them, making them whole again.
A broken clavicle usually requires a
simple arm sling to be worn for about six weeks. Children with broken
clavicles are often equipped with a figure of 8 clavicle strap that keeps
their clavicle immobilized until it heals, which is usually three to four
weeks. Most adults with the fracture will also use a figure of 8 splint or
strap and will probably sleep in a chair or in bed with extra pillows
because the fracture takes a week or two to get the healing process going
and it is important not to roll onto the bone while sleeping.
Your doctor will examine the fracture
site for neurovascular damage and take x-rays of the injured area, including
the joints above and below the primary injury site. He or she will ask for
details about how the injury occurred, and will need to know about any
previous accidents resulting in a fractured bone.
Healing is considered complete when
there is no motion at the fracture site and x-rays reveal complete bone
Patients with broken clavicles will
usually be able to exercise their shoulders after three weeks of
Golfers can expect to miss the walk
behind the ball for up to 12 weeks.
Prostate Cancer in the news again
Every week some male asks me about the
PSA test, and even knowing exactly what their PSA numbers have been for the
past 10 years. You think I joke? I do not.
The male child is born with a
subconscious fascination for the anatomy he finds in his nappy. Watch an
infant learning this. As the boy turns into man, what goes on in his
underpants becomes a major pre-occupation. As the man turns older, fear of
cancer in the underpants is an even greater pre-occupation.
Unfortunately prostate problems are
extremely common, a situation we men have to live with. Like all things,
there is a downside as well as the fun side. In fact this year in the United
States, almost 180,000 men will be told that they have prostate cancer.
With all our older friends getting
prostate problems, does this mean there is a rise in the incidence? Are our
underpants too tight? One reason for the ‘apparent’ increase is the fact
that prostate cancer is a disease of aging, and we are all living longer.
The statistics show that by age 50, almost 50 percent of American men will
have microscopic signs of prostate cancer. By age 75, almost 75 percent of
men will have some cancerous changes in their prostate glands. Do the maths.
By 100 we’ve all got it!
So does this mean that life really ends
at around 76? Fortunately no. Most of these cancers stay within the
prostate, producing no signs or symptoms, or are so slow-growing, that they
never become a serious threat to health. The good news is you die of
something else before the prostate gets you! You die with it, rather than
The real situation is that a much
smaller number of men will actually be treated for prostate cancer. About 16
percent of American men will be diagnosed with prostate cancer during their
lives; 8 percent will develop significant symptoms; but only 3 percent will
die of the disease. Put another much more positive way, 97 percent won’t die
from prostate cancer.
While prostate cancer can be
‘aggressive’, breaking out from the prostate gland itself and attacking
other tissues, including brain and bone, fortunately this is the minority
scenario. The great majority of prostate cancers are slow growing, and it
can be decades between the early diagnosis and the cancer growing large
enough to produce symptoms.
So let’s look at diagnosis and get the
“blood test” out of the way first. The blood test is called Prostate
Specific Antigen, or PSA for short (we medico’s love acronyms). Up till then
we had another test called DRE (digital rectal examination), which, quite
frankly, was not all that popular. So when news came through about a “blood
test”, millions of men began rejoicing and the sale of rubber gloves
plummeted. Unfortunately, PSA is not a go/no-go test. A normal range test
doesn’t guarantee you haven’t got it, and an elevated result doesn’t
automatically mean that you are about to claim early on your life insurance
(or your dependents, anyway).
However, there is good news. We are
becoming smarter with the PSA test. Serial PSA examinations can show the
rate of growth. This gives us ‘Staging’ with four main grades. Stage I
cannot be felt and is diagnosed through pathological testing. Stage II can
be felt, but it is confined to the prostate. Stage III is coming out of the
gland and Stage IV has grown into nearby tissues.
There is also the biopsy to contend
with. Sticking a needle into the prostate isn’t fun either, and in my
opinion is a bit hit and miss, but now there is the application of the MRI
to try to avoid biopsy.
This is where you need to discuss your
options with your doctor. If you are a young man with stage IV, then you
have to make up your mind quickly. But if you are 75 with stage I or II,
then you have more time, as you will most likely die of other causes rather
than prostate cancer. “Watch and Wait” has much going for it, but you must
be prepared to get to know your urologist. Pick a young one!
Living Wills – make one before you need it!
One medical situation that appears not
to be well understood, is the making of Living Wills. I am repeatedly asked
about whether a Living Will is legal in this country, and how do you enforce
the provisions. Read on, all will be made clear.
A couple of years ago, there was a
small paragraph in one of the Bangkok English language daily papers,
reporting on the fact that Living Wills were now accepted as being legal in
Thailand. I cheered as I read it. It was ‘about time’, in my opinion.
However, there is confusion in the
minds of many people, as to what a “Living Will” actually is and what it
covers. First off, it is not euthanasia. Borrowing from the Mayo Clinic in
the US, it states on their website: “This written, legal document spells out
the types of medical treatments and life-sustaining measures you do and
don’t want, such as mechanical breathing (respiration and ventilation), tube
feeding or resuscitation.” The important words to note are “life sustaining”
and “resuscitation”. Neither of these concepts imply medically assisted
Once again from the Mayo Clinic,
“Injury, illness and death aren’t easy subjects to talk about, but by
planning ahead you can ensure that you receive the type of medical care you
want, to take the burden off your family of trying to guess at what you’d
Remember that we are talking about
terminal situations here. Not situations from which it would be reasonably
expected that you will recover and still have a good quality of life. A
fractured hip when you are 90 is a serious situation, but provided you are
healthy otherwise, then it would be expected that you would recover. You
might need a stick for a while, but you would still be able to have a beer
with your mates or whatever your pursuits were before the incident. In other
words, the expectancy of a reasonable quality of life is there.
However, if you are in the terminal
phase of metastatic cancer, which has progressed despite treatment, the
future quality of life is not there. Artificially prolonging life under that
situation is then covered by the Living Will.
As an example, note the following:
The Living Will is made while in sound
mind. It is not something you scribble out while lying in God’s waiting
room. An example of a Living Will. “Being of sound mind and understanding
all the implications, I ask that this document be brought to the attention
of any medical facility in whose care I happen to be, and to any person who
may become responsible for my affairs.
“This is my ‘Living Will’ stating my
wishes in that my life should not be artificially prolonged, if this
sacrifices my Quality of Life.
“If, for any reason, I am diagnosed as
being in a terminal condition, I wish that my treatment be designed to keep
me comfortable and to relieve pain, and allow me to die as naturally as
possible, with as much dignity as can be maintained under the circumstances.
“As well as the situation in which I
have been diagnosed as being in a terminal condition, these instructions
will apply to situations of permanently unconscious states and irreversible
“In the case of a life-threatening
condition, in which I am unconscious or otherwise unable to express my
wishes, I hereby advise that I do not want to be kept alive on a life
support system, and I do not want resuscitation, nor do I authorize, or give
my consent to procedures being carried out which would compromise any
Quality of Life that I might expect in the future.
“I ask that you are sensitive to and
respectful of my wishes; and use the most appropriate measures that are
consistent with my choices and encompass alleviation of pain and other
physical symptoms; without attempting to prolong life.
Now those are only examples. The
Bangkok Hospital Pattaya has a pro forma Living Will, which is also repeated
in the Pattaya City Expats website, I believe.
The message is that a Living Will is
not euthanasia, and that you must lodge it, before you need it!
A Wayside Inn of Ill Repute
My anatomy teacher, Professor Max
Hickey, had a wicked sense of humor. An anatomical out pocket on the bowel
is called a diverticulum, which comes from the Latin meaning “a small
diversion from the normal path”; however, Max taught us that it was Latin
for “a wayside inn of ill repute”. I certainly like Max’s definition better,
and his words have stayed with me for many years!
Inflammation of these anatomical out
pockets is called Diverticulitis, and the little pockets are called
‘diverticulae’ (Latin plural of diverticulum). So, the condition of having
diverticulae is called Diverticulosis, and if they become inflamed (from any
reason) this is called Diverticulitis.
Diverticulae are more common in
industrialized countries than in third world countries. The reason given for
this is the lack of bulk present in the diet of industrialized countries
allowing muscle contractions to create localized areas of high pressure
allowing diverticulae to form, popping through the lining of the bowel.
The prevalence of diverticulae clearly
increases with age. While fairly uncommon during the first four decades of
life they reach a frequency of 50 percent in people older than 65. And
welcome to the wonderful life of a retiree.
It must be remembered that
Diverticulosis has no symptoms, but Diverticulitis does when they can
rupture into the abdominal cavity, cause localized irritation and
inflammation or produce an abscess. This is called acute diverticulitis.
Patients who have diverticulitis can
present with a rather sudden onset of pain located in the lower left part of
the abdomen over the sigmoid colon. It frequently is exquisitely tender and
is associated with fever and a high white blood cell count.
Secondly, they can painlessly start to
have significant amounts of rectal bleeding. When diverticulae bleed it is
usually rather large amounts 500 ml or more. This happens without any
inflammation whatsoever. The cause is a weakening of the blood vessel
adjacent to the diverticulum.
Acute diverticulitis can be diagnosed
by a typical history and a physical exam showing tenderness over the sigmoid
colon which is located in the left lower part of the abdomen. If fever and a
high white blood cell count are present this is confirmatory. A CAT scan or
ultrasound of the lower abdomen can be very helpful in showing an
inflammatory mass over the sigmoid colon.
Diverticular bleeding can be a bit more
difficult to diagnose and is frequently a "diagnosis of exclusion".
Fortunately this is not common. Less than 5 percent of people with
diverticular disease of the colon will bleed.
Acute diverticulitis is treated with
antibiotics for 7-10 days. These antibiotics frequently have to be given
intravenously. Diet is often severely limited during the first few days of
treatment. Most patients will recover completely, but occasionally surgery
is necessary in order to drain all the infected material and completely
empty an abscess cavity. At times this can require the creation of a
colostomy to remove the feces from the infected area. After this has healed
(usually about 6 weeks) the colostomy is removed and the colon is restored
to its original state with removal of the diseased portion of the colon.
Bleeding diverticulosis is managed
initially by monitoring the patient closely regarding his rate of blood loss
and giving blood transfusions if necessary. Fortunately the bleeding
normally stops. If not, the part of the colon containing the bleeding
diverticulum needs to be surgically removed.
There is much written but little proof
that anything can be done to prevent a recurrence of bleeding diverticular
disease of the colon or acute diverticulitis short of a surgical resection.
Of those that have bled about 15 percent will have a second bleed. If a
second bleed occurs, the risks increase to 50 percent they will have a
third. About 25 percent of those patients with acute diverticulitis will
have a relapse and many of these will need a surgical resection. The use of
a high fiber diet or use of stool softeners has been advocated to prevent
recurrences of this disease by some researchers. The theory is that bulk in
the colon in the form of a high fiber diet will help prevent recurrences by
preventing localize high pressures from occurring. I remain unconvinced.
Dengue Fever - now we have a vaccine!
Many people were under the impression
that Dengue is a mild illness, caused by a mosquito bite. However, with the
Thai TV actor ending up in intensive care with Dengue Hemorrhagic Fever,
this has brought home the realities of this disease.
The female Aedes aegypti mosquito,
which carries the Dengue virus, is the culprit. Dengue fever is endemic
locally and the change in the weather is partly to blame (not to be confused
with “climate change”, by the way).
Since the beginning of this year,
dengue fever is reported to have been seen in 14,000 people. The Disease
Control Department says the rainy season each year is usually the breeding
period for the dengue virus, and it has certainly rained recently.
So, despite my previous pleas (and
those of the Public Health Department), Dengue Fever and its potentially
fatal variant, Dengue Hemorrhagic Fever (DHF) is still with us. The latest
information has now prompted me to repeat my advice on this subject. If you
remember reading about it before, I apologize, but the subject matter is
very important. This is an important ailment, which can be avoided.
However, first you should understand a
little more about Dengue and its history. It was first described in 1780 by
a Benjamin Rush in Philadelphia (so it didn’t start here) when the name
Break Bone Fever was applied, with the symptoms of pain in the bones and
rise in temperature. The name “Dengue” came in 1828 during an epidemic in
Cuba. The new name was a Spanish attempt at a Swahili phrase “ki denga pepo”
which describes a sudden cramping seizure caused by an evil spirit! Let me
assure you that the local brand of Dengue Fever owes nothing to spirits,
evil, bottled or otherwise.
The dengue virus is related to Japanese
encephalitis, Murray Valley encephalitis and Yellow fever, and there are
four “serotypes” or subgroups of it.
The mosquito lays its eggs in water
containers, preferring the clean water found in water tanks and pots, in the
saucers under pot plants and even under the pet’s food dish. Inside
discarded car tyres is another favorite spot. These mosquitoes are not of
the adventurous type and feed during the day and spend their time within 200
meters of their hatchery. Consequently, the eradication of any local
breeding areas becomes very important towards maintaining your own health,
as you can see. Keep your home free from lying water for a radius of 200
meters and you’re looking good!
Simple Dengue (if you can call it that)
has an incubation period of around four to seven days and then the full
blown symptoms of high fever and headache begin. The headache is usually
behind the eyes and is made worse by eye movement. From there the pains
progress to the limbs with acute muscle pains, which gave it the old name
“Break Bone Fever”.
On the other hand, Dengue Hemorrhagic
Fever (DHF) can certainly be fatal! It appears that Serotype 2 may be the
culprit here, but does not usually produce DHF unless you have been
previously bitten by types 1, 3 or 4. In addition to the symptoms of
Classical Dengue the skin begins to bruise very easily as the blood
hemorrhages into the skin. Children are generally more susceptible to this
than adults. This also becomes much more of an emergency and is best treated
in the Intensive Care Unit (ICU) of your favorite hospital.
With our ability to treat the viral
ailments being very limited, the defense against the Dengue virus used to
lie in the preventive measures. The other precautions are to wear long
trousers and long sleeved shirts, especially at sun up and sun down, when
the mosquito is at its most ravenous. The other factor to remember is “D”
for Dengue and “D” for DEET. DEET is the magic ingredient in mosquito
repellents, so when you go to buy some, check the label – if it has DEET,
then get it. However, there is also now a vaccine against Dengue (ages
9-45), available at the Bangkok Hospital Pattaya till February 28. B. 9,600
for three injections. Looks worthwhile to me.
Good Exercise. Does that include sexercise?
The new check-up programs are out in my
hospital with all the discounted items so you can chart your health (or
otherwise) and star remedial action.
Probably the commonest advice a doctor
gives to patients who have just done a check-up is to lose weight and get
some exercise. Does that ring a bell in your memory? Was that part of the
advice after your annual physical check-up?
Unfortunately, there seems to be very
little real understanding of what exercise should consist of, how often,
what type, how long and what about sex? For example, I was reading an
article on exercise the other day and it said authoritatively that one
should wear comfortable clothing and socks with the correct size of
non-slippery, shock-absorbing shoes. If this includes sexercise, there are
some strange shoe fetishes out there that I haven’t heard of yet!
However, getting a little serious,
exercise will be good for you, provided that you pick a form of exercise
that is not harmful for you! Now I know that looks as if I have put my money
on every horse in the race, but take that sentence at its face value.
Enough research has been done to show
that regular exercise is beneficial for everybody, in both the physical and
psychological aspects, but, and it is a big ‘but’, all forms of exercise
have relative bodily risks, and this has to be taken into account before you
buy a pair of expensive jogging shoes and tackle a 10 km trot up Pratamnak
Hill in the middle of the day.
True stories – a medical colleague in
Australia took up playing squash when he turned 50 to improve his fitness
and dropped dead on the court of a heart attack, and another acquaintance of
mine turned 40, decided he wasn’t fit, bought a bicycle to ride to work each
day and was run over by a bus.
The same article that advised
non-slippery shoes, did have some wise words, however. These included to
choose appropriate exercise according to your ability. Never exceed your
limit. Remember that it is not the harder the better. If you have acute
medical problems (such as fever, or pain), stop exercising. If you have
chronic medical conditions (such as hypertension, diabetes, ischemic heart
disease and arthritis), seek advice from your doctor or physiotherapist
All of these I agree with. Remember
that if you are happy to take your body to your medical advisor when it is
sick, take it back to your doctor for advice on how to keep it fit as well.
The other words of wisdom in the
article suggested that for prolonged exercise such as hiking, continually
drink water to supplement the loss of body fluid due to sweating. Do not
wait until you are thirsty. Take appropriate breaks during exercise. Do not
over-exert yourself. Forget about “powering through the pain barrier”. Leave
that for Tour de France cyclists and their “special” hormones.
As well as the form of exercise, there
is the frequency. At least three times per week is recommended and 20-30
minutes (or more) is necessary each time, to derive the maximum benefit. And
always remember, if there is dizziness, fainting, shortness of breath, chest
pain, vomiting, nausea or severe pain during exercise, stop exercising
immediately and seek medical advice as soon as possible.
Now I did mention at the start of this
week’s article, the word “sexercise”, and some of you have been impatiently
reading, while nervously fiddling with your expensive packet of Viagras,
Kanagras, Cialis, Sidegra and other lead-in-your-pencil medications (I draw
the line at tiger willy). OK, what about sex? The advisability of this form
of exercise when you have some chronic complaint (such as hypertension,
diabetes, ischemic heart disease, etc.), should be part of the advice you
get from your doctor beforehand. The danger of over the counter willy
stiffeners is that you don’t get advice with them and the strain of sexual
activity may not be the best for you.
Finally, the learned article did say
“Exercise with friends. Company provides enjoyment, mutual encouragement and
support.” That goes for sexercise too I would imagine, but take the joggers
The Seven Deadly Sins!
Heart disease is still one of the
greatest killers of mankind, ranking a strong second after the Songkran road
toll – OK, I exaggerated that a little, but I still maintain that any
celebration in which 500 people get killed is a blight on the face of our
Interestingly, if you look at the major
causes of death by development of the countries, in the high income
countries death toll comes from heart disease, stroke and lung cancer,
middle income countries have stroke, heart disease and lung disease and for
the low income countries it is heart disease, respiratory infections and
There are many reasons for the
differences, including dietary, socio-economic, educational, development of
health services and tobacco and alcohol abuse. However, this week I am only
going to address heart disease, and the seven deadly ‘sins’ that can predict
your likelihood of getting (and suffering from) heart disease.
1. High Blood Pressure: 20 percent of
elderly people suffer from this condition. Imagine trying to blow through a
long tube. If the tube becomes constricted for any reason, you will have to
blow harder, increasing the pressure. Blood pressure is the same – if the
arteries are constricted or less pliable, it takes a greater pressure to
force the blood around. The heart has to work harder to produce the
increased pressure, and eventually the heart gets tired and fails.
2. High cholesterol: High cholesterol
foods such as egg yolk, offal, animal brain, animal fats, dairy products,
seafood, oyster, squid, etc., leave deposits in the blood vessel walls. As a
result, the fat “plaque” on the vessel walls obstructs the blood flow and
this will eventually cause heart disease, as per the first deadly sin above.
3. Smoking: Smoking is a primary factor
in the causation of coronary artery disease. Smokers are at a much higher
risk, even two times more than non-smokers. Smoking increases adrenaline,
which causes an increased heart rate, increased blood pressure and lowers
the amount of oxygen carried by the blood.
4. Diabetes: Diabetic people have twice
the risk of congestive heart failure than people with normal blood sugar
levels, due to their increased weight (see number 5) and high cholesterol
levels produced in the blood.
5. Obesity: People who are fatter than
average have to face a 30-40 percent increase in risk of heart disease
compared to thin people. In just carting around the extra weight, the heart
has to work harder (and the knee and hip joints wear out). Try walking
around with 10 kg extra on your back and tell me how you feel at the end of
a week. Tired? Of course. And your heart is tired too.
6. Stress: Stress is not totally bad
for us, as it keeps you going, and in an appropriate level actually
stimulates our curiosity and motivation; however, by getting over-stressed,
adrenaline levels are increased and this may lead to abnormal heart
function. Though stress is not the main cause of heart disease, it can make
the artery walls less flexible, which is the beginning of heart disease.
7. Lack of exercise: Exercise is the
best way to increase high density lipoprotein (HDL) – “good” cholesterol
that prevents the arteries becoming abnormal. By exercising, blood pressure,
body weight, and the possibility of thrombosis will be reduced.
So you can see just how these seven
risk factors all are inter-related. Keep the arteries clean, watch the diet
and ensure you have a reasonable level of exercise. Check the Blood Pressure
and stop smoking.
Remember too, that as you get older,
the chances (and risks) of heart disease are higher. (Young males are more
likely to die from Songkran effects than heart failure.) Statistically, men
aged over 40 years as well as the postmenopausal women have a higher risk
Despite our knowledge, we still cannot
predict exactly when the demise will come, but looking at the big picture,
we do know that smoking, overweight, unfit diabetics with high blood
pressure and high cholesterol do not live as long as non-smoking, fit, lean
people with normal blood pressure, and normal blood sugar and cholesterol
Do you know your levels? A brief
medical check-up will tell you.
A PET scan is not
an X-Ray of your dog
There is only December
30 and 31 left in which to pay to get the discounted Check-up Packages in my
hospital. However, as long as you have paid before December 31, you have
till February 28 to actually have the physical examination.
The usual request is “I
want everything.” And by that they mean blood testing. If I told them that
sitting on my desk is the “Manual of Use and Interpretation of Pathology
Tests” which is almost 400 pages and there are about five tests per page.
Imagine the bill for all that lot! But I doubt if many of you need Basement
Membrane Antibodies to be done for any reason.
There is also, in the
collective subconscious, interest in a “whole body scan” which is thought of
as some magical device that you can walk into in one end and out the other
and a print-out will tell you (and us) exactly how you are inside and out.
Every organ! Even Willy the Wonder Wand! Unfortunately, this is stretching
the truth somewhat. Machines like that are only seen in Star Trek movies.
However, there is the
PET scan, which is a specialized form of whole body scanner, that can give
an indication of what is going on inside.
PET stands for Positron
Emission Tomography and is a type of nuclear medicine imaging. Nuclear
medicine is a subspecialty within the field of radiology that uses very
small amounts of radioactive material to diagnose or treat disease and other
abnormalities within the body.
imaging procedures are noninvasive and usually painless medical tests that
help physicians diagnose medical conditions. To be able to produce the
images in a PET scan, you have to have radioactive materials, called a
radiopharmaceutical or radiotracer, and these are injected into your veins.
The radioactive material has a very short life and is usable for only about
two hours, though it will take a day before you have excreted it all.
The radioactive energy
is detected by a device called a gamma camera, a (positron emission
tomography) PET scanner. These radiology devices work together with a
computer to measure the amount of radiotracer absorbed by your body and to
produce special pictures offering details on both the structure and function
of organs and other internal body parts.
The PET scanner is most
usually used in cancer medicine and can demonstrate a ‘hot spot’ to show up
the primary cancer, stage a cancer, show any metastases (spread), and even
show whether cancer treatment modalities are working. For example, the PET
scan can show the difference between scar tissue and active cancer tissue.
The benefits provided
by PET scans are primarily because the information provided by nuclear
medicine examinations is unique and often unattainable using other imaging
For many diseases,
nuclear medicine scans yield the most useful information needed to make a
diagnosis or to determine appropriate treatment, if any.
Nuclear medicine is
much less traumatic than exploratory surgery.
By identifying changes
in the body at the cellular level, PET imaging may detect the early onset of
disease before it is evident on other imaging tests such as CT or MRI.
The risks are very low.
Because the doses of radiotracer administered are small, diagnostic nuclear
medicine procedures result in minimal radiation exposure. Thus, the
radiation risk is very low compared with the potential benefits.
Nuclear medicine has
been used for more than five decades, and there are no known long-term
adverse effects from such low-dose exposure.
Allergic reactions to
radiopharmaceuticals may occur but are extremely rare.
Injection of the
radiotracer may cause slight pain and redness which should rapidly resolve.
Women should always
inform their physician or radiology technologist if there is any possibility
that they are pregnant or if they are breastfeeding their baby.
Can you get this kind
of scan here? Yes, at Wattanosoth Hospital in Bangkok, and it costs around
60,000 baht last time I asked. However, if you purchase a Chivawattana
personal health insurance card (available at Bangkok Hospital Pattaya) it
has a discount for the PET scan (as well as many other benefits). Well
worthwhile looking into if a PET is on the agenda.
Christmas Disease – it’s not Happy Holiday disease!
has nothing to do with Happy Holidays, Christianity, or Santa, mangers,
three wise men and a bottle of myrrh. But it has everything to do with
Stephen. And not St. Stephen but Stephen Christmas, that is.
Stephen, a young
British lad, was the first patient with a bleeding tendency recognized
to have a different form from “classical” hemophilia (or haemophilia if
you come from the right hand side of the Atlantic Ocean).
His condition was
studied by researchers Biggs, Douglas, and Macfarlane in 1952, who
discovered that young Stephen was missing a different coagulation factor
than the more usual one (which is known as Factor VIII). They named
Stephen’s missing factor as Factor IX, and his condition became known as
Just to confuse the
issue, we also call Christmas Disease by other names, including Factor
IX deficiency, hemophilia II, hemophilia B, hemophiloid state C,
hereditary plasma thromboplastin component deficiency, plasma
thromboplastin component deficiency, and plasma thromboplastin factor-B
deficiency. There’s probably more, but Christmas Disease has a much
nicer “ring” to it. (Probably “Jingle Bells” at this time of year!)
From the diagnostic
viewpoint, it is very difficult to differentiate between classical
hemophilia (my editor comes from the left hand side of the Atlantic, so
it is spelled with “e”) and Christmas Disease. The symptoms are the
same, with excessive bleeding seen by recurrent nosebleeds, bruising,
spontaneous bleeding, bleeding into joints and associated pain and
swelling, gastrointestinal tract and urinary tract hemorrhage producing
blood in the urine or stool, prolonged bleeding from cuts, tooth
extraction, and surgery and excessive bleeding following circumcision.
covers around one in seven cases of the total hemophilia incidence and
is around 1/30,000 in the general population. This disease is also male
dominated, being called a sex-linked recessive trait passed on by female
carriers. This means the bleeding disorder is carried on the X
chromosome. Males being of XY make-up will have the disease if the X
they inherit has the gene. Females, who have XX chromosomes, are only
carriers if either X has the bleeding gene.
Hemophilia has been
noted in history for many years, and Jewish texts of the second century
A.D. refer to boys who bled to death after circumcision, and the Arab
physician Albucasis (1013-1106) also described males in one family dying
after minor injuries.
In more recent
history, royal watchers know that Queen Victoria of Britain’s son
Leopold had hemophilia, and that two of her daughters, Alice and
Beatrice, were carriers of the gene. Through them, hemophilia was passed
to the royal families in Spain and Russia, leading to one of the most
famous young men with the disease, Tsar Nicholas II’s only son Alexei.
In the 1800’s
physicians thought that the bleeding occurred because of a structural
problem in blood vessels. In 1937 a substance was found in normal blood
that would make hemophilic blood clot, which was named “anti-hemophilic
In 1944 researchers
found in one case that when the blood from two different hemophiliacs
was mixed, both were able to clot. Nobody could explain this until 1952,
when the researchers in England realized there were two types of
hemophilia. They called his version hemophilia B, or “Christmas
disease,” and the more prevalent kind hemophilia A, or “classic
With the discovery
of A and B types came the realization that there must be different types
of “anti-hemophilic globulin” involved in the clotting process. Names
were assigned to these various “coagulation factors” by an international
committee in 1962. Hemophilia A is a deficiency of Factor VIII, and
hemophilia B is a deficiency of Factor IX.
Once it became
clear that hemophilia was caused by a deficiency of a coagulation
factor, replacement of the missing factor became the method of
treatment. In the early 1950’s animal plasma was used. By the 1970’s,
coagulation factor concentrates made from human plasma were available,
and by the 1980’s we could guarantee that it was HIV free. It has been a
long road since Stephen Christmas.
check-up packages at Bangkok Hospital Pattaya have to be paid for by
December 31, but you can delay having the test up till 28 February 2017.
Where’s my readers?
I don’t need
these any more!
By “readers” I don’t mean those kind
souls who read my columns, but I am referring to reading glasses. I just
opened my top drawer at work and there were eight pairs of readers! No, it
wasn’t a “Buy 1 and get 7 free!” These were the result of wear and tear,
broken side pieces, lenses missing, broken frames, unsuccessful repairs with
Super Glue or Araldite (the Greek Goddess of stickiness) and the list goes
on. I did, however, manage to successfully glue my finger to one lens with
Super Glue. It did neither the lens, or my finger, any good at all.
A little history here, which will
probably remind you of your time of decreasing visual acuity.
26 years ago I was ready to admit that
my near vision was gone. Reading a map was just not possible, even holding
the map in front of the headlights. A visit to the optometrist saw me
leaving with suitable contact lenses which introduced a whole new world to
my life. I could read the destination signs on busses and street signs
Unfortunately this Utopia was not to
last. Stronger prescriptions for the contact lenses did help, but were not
the answer. Remembering to remove the contacts every night was a bit hit and
miss, but I always knew in the morning as the lenses were stuck to my
eyeballs and everything was blurred.
I did try and remember to use a sterile
technique putting the lenses in, but that too would fail regularly and I
would have to do without, which suggested to me that a white stick and a
Labrador was next. Of course there were also the dropped lenses with me on
all fours looking for the errant bit of soft plastic.
I struggled on, but then found that my
distance vision was not as good as it used to be. Recognizing faces across
the street was difficult. By now I had arrived at a situation where driving
at night was taking my life in my hands.
It was at this point that I discussed
my vision with Dr. Somchai Trakool Choke-satian in the SuperSight surgery
department at the Bangkok Hospital Pattaya.
It was at that initial consultation
that I discovered that SuperSight was not just changing the lens in the eye,
but a thorough eye examination to ensure that the eye itself is healthy,
other than the cataracts and hardening of the natural lens. This examination
takes about one hour, so it’s not a case of “Read the bottom line,” and it’s
all over style exam.
Dr Somchai advised me that my eyes were
suitable for the SuperSight lenses and then went on to discuss all the pros
and cons of the surgery and the final results that could be expected, and
then told me to go and think about it.
I did that by speaking to all the
people I knew who had SuperSight surgery done before. To a man the answers
were the same, “Wish I’d done it years ago”.
So a convenient date was selected for
Dr. Somchai and myself and the stage was set. No turning back.
I am asked by others whether the
operation was painful and I can honestly say it was not. It is a weird
sensation having someone ‘inside’ your eyeball, but not painful. Under local
anesthetic I found I could relax and keep my eye still as Dr. Somchai did
his magic. And the results are magical. And the freedom from the glasses is
Now two months after the operation, I
still pat my pocket for the readers as I sit down in front of the computer
and then realize I don’t need them. And as a funny ending, I can now wear
T-shirts without a pocket for the glasses. My wardrobe has doubled! And I
can finally read the numbers on the remote for the TV.
For more information contact the
SuperSight surgery department at the hospital, they are nice people.
Do you have “sugar”
Diabetes is a serious ailment,
which can arise for many reasons, and can affect many systems in the
human body. Diabetes, often called “sugar” by patients, is diagnosed and
monitored mainly through a simple blood test – the Blood Glucose level.
Glucose is a type of sugar found in
fruits and many other foods (this includes lactose and fructose). It is
the main source of energy used by the body. Most of the carbohydrates
that people eat are also turned into glucose, which can be used for
energy or stored in the liver and kidneys as glycogen.
To stop the sugar levels just
increasing daily, a balance is achieved through a hormone called Insulin
which helps the body use and control the amount of glucose in the blood.
Insulin is produced in areas of the pancreas called ‘islets’ and
released into the blood when the level of glucose in the blood rises. In
simple terms, people who do not produce enough insulin develop diabetes.
People can also develop diabetes if they do not respond normally to the
insulin their bodies produce. This occurs most commonly when a person is
overweight, and since obesity is on the rise, so are various types of
Normally, blood glucose levels
increase slightly after a person eats a meal. This increase causes the
pancreas to release insulin so that blood glucose levels do not get too
high. Blood glucose levels that remain high over time can cause damage
to the eyes, kidneys, nerves, and blood vessels, which explains why good
glucose control is important.
There are many ways to carry out
blood glucose tests, including fasting blood sugar (FBS). This is a
measurement of blood glucose after fasting for 12 to 14 hours. For an
accurate fasting blood sugar test, do not eat or drink for 12 to 14
hours before the blood sample is taken. However, water can be freely
taken, as otherwise hemoconcentration occurs to give a falsely high
reading. This is often the first test done to detect diabetes, and
explains why fasting blood tests are usually done when having a medical
The other common test is called the
random blood sugar (RBS). A random blood sugar measurement may also be
called a casual blood glucose test. This is a measurement of blood
glucose that is taken regardless of when the person last ate a meal.
Sometimes several random measurements are taken throughout a day. Random
testing is useful because glucose levels in healthy people do not vary
widely throughout the day, so wild swings may indicate a metabolic
Glucose tolerance testing can also
be done, usually to confirm a condition known as gestational diabetes,
which can occur during pregnancy. An oral glucose tolerance test is
simply a series of blood glucose measurements taken after a person
drinks a liquid containing a specific amount of glucose; however, this
test is not used to diagnose diabetes.
To monitor the treatment of
diabetes, there are another couple of tests which can be carried out.
The commonest is Glycated Hemoglobin, otherwise referred to as HbA1c.
This test actually is an indicator of the average glucose concentration
over the life of the red blood cells (which is taken as over the
previous three months).
Another is the Serum C-Peptide
which is used to investigate low blood sugar levels, done by measuring
the C-Peptide which is produced by the Beta cells in the pancreas.
“Normal” levels may vary from lab
to lab, but generally the range taken for FBS is that the level should
be less than 110 milligrams per deciliter (mg/dL).
Diagnosis of diabetes needs a
fasting blood glucose level higher than 125 mg/dL on two separate days.
A fasting glucose level below 40
mg/dL in women or below 50 mg/dL in men that is accompanied by symptoms
of hypoglycemia (low blood sugar) may indicate an insulinoma, a tumor
that produces abnormally high amounts of insulin. Lower than expected
glucose levels can also indicate Addison’s disease, an underactive
thyroid gland or pituitary gland, liver disease (such as cirrhosis),
malnutrition, or a problem that prevents the intestines from absorbing
the nutrients in food.
So you can see “sugar” is important
which is why we have specialist endocrinologists at my hospital.
where are you now?
Bernardino Ramazzini (1633 – 1714) was an Italian physician
who is considered to be the founding father a rather different medical
specialty called Occupational Medicine. One of the lesser known medical
specialties, this is the study of worker health, how the workplace affects
health, the man-machine interface, industrial exposure to contaminants and
many other occupational hazards. (This is not something very well known in
There are many medical
conditions caused by work, right the way from Housemaid’s Knee and another
example of occupationally induced conditions is ‘Vibration White Fingers’
and comes under the general umbrella of an interesting set of conditions
known as Raynaud’s phenomenon.
Since doctors like to
have conditions named after them, Raynaud’s phenomenon comes from Dr.
Maurice Raynaud, a French physician who published a report in 1862 of a
young woman whose fingertips changed colors when she was cold or under
stress. He is credited with the discovery of the condition.
sometimes called Raynaud’s syndrome or disease, is a disorder of blood
circulation in the fingers. This condition is usually produced by exposure
to cold which reduces blood circulation causing the fingers to become pale,
waxy-white or purple. This condition is sometimes called “white finger,”
“wax finger” or “dead finger”. These attacks occur when the hands or the
whole body get cold either at work or at home. Household or leisure
activities resulting in cold exposure can include washing a car, holding a
cold steering wheel, or the cold handlebars of a bicycle. Attacks of white
finger can also occur when a person is outdoors watching sports, or while
gardening, fishing or golfing in cold weather.
Typical attacks occur
with tingling and slight loss of feeling or numbness in the fingers,
blanching or whitening of the fingers, usually without affecting the thumb,
and pain, sometimes with redness, which accompanies the return of blood
circulation generally after 30 minutes to two hours.
Many cases of Raynaud’s
phenomenon are such that we cannot identify the cause. To escape the
embarrassment of admitting that we just don’t know, we call this “primary
Raynaud’s phenomenon” or even “constitutional” white finger. However, when
we do know the occupational cause of Raynaud’s phenomenon we call it
“secondary Raynaud’s phenomenon”!
In the occupational
sphere, there are many causes of this secondary condition. It is most
commonly associated with hand-arm vibration syndrome but it is also involved
in other occupational diseases. Awareness of the condition can help prevent
the disorder from occurring or progressing, as if not detected in the early
stages, the disorder can permanently impair blood circulation in the
phenomenon is not life threatening, severe cases cause disability and may
force workers to leave their jobs and workman’s compensation issues may end
up in courts of law. Although rare, severe cases can lead to breakdown of
the skin and gangrene. Less severely affected workers sometimes have to
change their social activities and work habits to avoid attacks of white
The underlying cause
relates to the physiology of maintaining an even body temperature. Usually,
the body conserves heat by reducing blood circulation to the extremities,
particularly the hands and feet. This response uses a complex system of
nerves and muscles to control blood flow through the smallest blood vessels
in the skin. In people with Raynaud’s phenomenon, this control system
becomes too sensitive to cold and greatly reduces blood flow in the fingers.
Exposure to vibration
from power tools is by far the greatest concern in secondary Raynauds.
Hand-held power tools such as chain saws, jackhammers and pneumatic rock
drillers and chippers can cause “hand-arm vibration syndrome”. This disorder
is the “vibration white finger”, “hand-arm vibration syndrome (HAVS)”, or
“secondary Raynaud’s phenomenon of occupational origin.” How many times have
you seen Thai construction workers with the flip-flop “safety” footwear
blasting away on concrete floors? Many times I am sure.
induced Raynaud’s phenomenon occurred in the early years, before the
cancer-causing effects of vinyl chloride monomer were known. Workers exposed
to high levels of this chemical also experienced Raynaud’s phenomenon.
So that is the story of
Raynaud’s phenomenon. Fortunately, in our warm tropical climate it is not
seen too often, other than the occupational secondary variety.